WebbBackground and objective: Apical thinning is a well-known phenomenon in myocardial perfusion SPECT, often attributed to reduced myocardial thickness at the apex of the left ventricle. Attenuation correction processing appears to exaggerate this effect. Although currently there is agreement that reduced apical counts are not a diagnostic indicator, … WebbNew, small, mild reversible perfusion defect in the anterolateral apex involving less than 5% of the LV myocardium. 2. Gated post-stress SPECT calculation of left ventricular ejection fraction is 60%, without evidence of focal left ventricular wall motion or wall thickening abnormalities.. Doctor's Assistant: The Doctor can help. Just a couple quick …
Small, mild reversible perfusion defect in the anterior
Webb2 maj 2024 · Left bundle branch block (LBBB) is usually an acquired conduction defect with the prevalence increasing with age. 1, 2, 3 It occurs as a sequela of coronary artery disease (CAD), congenital heart disease, valvular heart disease, cardiomyopathy, myocarditis, and can also be iatrogenic or degenerative in nature. 4, 5 In patients with LBBB, the … WebbWhat isa a small reversible defect invovling the apical anterior wall, that is consistant with possible ischemia this person did have open heart sugery in the past 6 yrs and has had … first oriental market winter haven menu
Reversible myocardial perfusion defects in patients not ... - PubMed
Webb1 juli 2012 · Mine was also in the anterior wall, and my cardiologist said it was probably an artifact. It's a little harder (for me, anyway) to understand how a reversible defect could be an attenuation artifact, but today I saw a second cardiologist who settled the question (to his and my satisfaction, anyhow) with a stress echocardiogram, done in his office. WebbYes, in general a reversible defect is considered to be ischemic. However, it needs to be placed in the patients clinical context. It may not be clinically significant if the patient is … Webb1 sep. 2013 · The sample size was chosen by conservatively estimating that 5% of individuals undergoing a cardiac perfusion study at our institution had a true focal perfusion defect. We calculated the sample size necessary to achieve a standard power of 0.8 for a detectable change of 2.5% from any correlate (single tail with α = 0.05). first osage baptist church